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Zinc aspartate suppresses proliferation and Th1/Th2/Th17 cytokine production of pre-activated human T cells in vitro
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Lower serum zinc levels in patients with multiple sclerosis compared to healthy controls
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Combined treatment with zinc aspartate and intravenous immunoglobulins (IVIGs) ameliorates experimental autoimmune encephalomyelitis (EAE)
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Antibodies against glycoprotein 2 display diagnostic advantages over ASCA in distinguishing CD from intestinal tuberculosis and intestinal Behçet's disease
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Gastric cancer in autoimmune gastritis - a casecontrol study from the Star Project on Gastric Cancer Research
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Magenkarzinom auf dem Boden einer Autoimmungastritis - eine Fall-Kontroll-Studie aus dem staR (gastric cancer research)-Konsortium
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Letter - multivariate clinical model for eosinophilic oesophagitis is this generalisable to a general population? : Authors reply
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Der neuroimmunologische Boomerang nach Herpes-Simplex-Enzephalitis
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Impact of in vivo high-field-strength and ultra-high-field-strength MR imaging on DNA double-strand-break formation in human lymphocytes
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Serological diagnosis and prognosis of severe acute pancreatitis by analysis of serum glycoprotein 2
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Zink - Bedeutung in der ärztlichen Praxis
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Inhibitors of dipeptidyl peptidase IV and aminopeptidase N target major pathogenetic steps in acne initiation
In: The journal of investigative dermatology. - New York, NY : Nature Publishing Group, Bd. 127.2007, 5, S. 1042-1051

Immune peptides related to dipeptidyl aminopeptidase IV/CD26
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Dipeptidyl aminopeptidases - basic science and clinical applications
In: Advances in experimental medicine and biology; 575

Triggering endogenous immunosuppressive mechanisms by combined targeting of Dipeptidyl peptidase IV (DPIV/CD26) and Aminopeptidase N (APN/ CD13) - a novel approach for the treatment of inflammatory bowel disease
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Normal T-cell development and immune functions in TRIM-deficient mice
In: Molecular and cellular biology . - Washington, DC : American Society for Microbiology, Bd. 26.2006, 9, S. 3639-3648

Dipeptidyl peptidase IV (DP IV, CD26) and aminopeptidase N (APN, CD13) as regulators of T cell function and targets of immunotherapy in CNS inflammation
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Increased concentrations of transforming growth factor [beta]1 and [beta]2 in the plasma of patients with glioblastoma
In: Journal of neuro-oncology . - Dordrecht : Springer, ISSN 0167-594x, Bd. 79.2006, 1, S. 61-65

Attractin, a dipeptidyl peptidase IV/CD26-like enzyme, is expressed on human peripheral blood monocytes and potentially influences monocyte function
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Dipeptidylpeptidase IV (DPIV) and alanyl-aminopeptidases (AAPs) as a new target complex for treatment of autoimmune and inflammatory diseases - proof of concept in a mouse model of colitis
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Dipeptidyl peptidase IV (DP IV, CD26) and aminopeptidase N (APN, CD13) as regulators of T cell function and targets of immunotherapy in CNS inflammation
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CD26/DP IV in T cell activation and autoimmunity
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Possible role of DP IV inhibitors in acne therapy
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Expression of dipeptidyl peptidase IV-like enzymes in human peripheral blood mononuclear cells
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Dipeptidyl peptidase IV regulates T cell function in CNS inflammation.
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Liver cirrhosis and "liver" diabetes mellitus are linked by zinc deficiency.
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The role of dipeptidyl peptidase IV (DP IV, CD26) in T cell activation and multiple sclerosis.
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Soluble vascular endothelial growth factor, soluble VEGF receptor Flt-1 and endothelial function in healthy smokers.
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The transmembrane adapter protein SIT regulates thymic development and peripheral T-cell functions.
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Regulation of in vitro and in vivo immune functions by the cytosolic adaptor protein SKAP-HOM.
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The imbalanced mixed lymphocyte reaction between maternal and fetal lymphocytes as well as between the lymphocytes of adult children and their parents is mediated by activated CD8+ T cells.
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Increased blood plasma concentrations of TGF-beta isoforms after treatment with intravenous immunoglobulins (IVIG) in patients with multiple sclerosis.
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Identification of extra- and intracellular alanyl aminopeptidases as new targets to modulate keratinocyte growth and differentiation.
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Dipeptidyl peptidase IV inhibitors with the N-terminal MXP sequence : structure-activity- relationships.
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Synergistic action of DPIV and APN in the regulation of T cell function.
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New results on the conformations of potent DP IV (CD26) inhibitors bearing the N-terminal MWP structural motif.
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Immunomodulatory effects of zinc.
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Specific inhibition of dipeptidyl peptidase IV (CD26) enzymatic activity - a new approach to psoriasis therapy?.
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Propolis and some of its constituents down-regulate DNA synthesis and inflammatory cytokine production but induce TGF-ß1 production of human immune cells.
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Different modes of dipeptidyl peptidase IV (CD26) inhibition by oligopeptides derived from the N-terminus of HIV-1 Tat indicate at least two inhibitor binding sites.
In: Eur. J. Biochem. 270(2003), S. 2147 - 2156

Changes of plasma concentrations of soluble vascular cell adhesion molecule-1 and vascular endothelial growth factor after increased perfusion of lower extremities in humans.
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CD26/DPIV in diseases of the central nervous system.
In: Langner, J. (Ed.) ; Ansorge, S. (Ed.): Ectopeptidases : CD13/aminopeptidase N and CD26/dipeptidylpeptidase IV in medicine and biology. New York [u.a.] : Kluwer Acad./Plenum Publ., 2002, S. 279 - 287

CD26/DPIV in hematopoietic cells - expression, function, regulation, clinical aspects.
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Calpains and cytokines in fibrillating human atria.
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Protein kinase C and ERK activation are required for TFF-peptide-stimulated bronchial epithelial cell migration and tumor necrosis factor-alpha-induced interleukin-6 (IL-6) and IL- 8 secretion.
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Arthus reaction to lepirudin, a new recombinant hirudin and delayed-type hypersensitivity to several heparins and heparinoids with tolerance to its intravenous administration.
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Inhibition of human leukocyte function, alanyl aminopeptidase (APN, CD13) and dipeptidylpeptidase IV (DP IV, CD26) enzymatic activities by aqueous extracts of Cistus incanus L. ssp. incanus.
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The role of dipeptidyl peptidase IV (DP IV) enzymatic activity in T cell activation and autoimmunity.
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Neutrophils synthesize and activate TGFß2.
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